Health Issue

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Health Issues

Your Name (First and Last)

Institution or School name

Annotated Bibliography

Barnes, P. J. (2016). Asthma-COPD overlap.Chest,149(1), 7-8.

The author Peter J. Barnes, from National Heart and Lung Institute, Imperial College London, London, England, performed research, and on the base of that research he found out mostly doctors can easily differentiate between COPD and asthma. Through his research, he also figured that the symptoms of asthma start off at a very young age, and responds to inhale therapy very well. There are specific allergies that are also associated with asthma. Contrary to asthma, COPD symptoms take time to develop. The response to inhaling therapy of a COPD patient is usually very poor. It develops by time and sometimes it is linked with chain smoking. However, both the diseases are different, yet, at times there can be patients who might have the symptoms of both COPD and Asthma. Such a condition is called asthma-COPD overlap syndrome ACOS. The author also states with regards to his research there can be a prediction of overlap due to asthma and COPD being common. Having said that, there is no such evidence that suggests that one illness protects the other ADDIN ZOTERO_ITEM CSL_CITATION citationIDa1o43f3lvol,propertiesformattedCitation(Barnes, 2016),plainCitation(Barnes, 2016),citationItemsid174,urishttp//zotero.org/users/local/zTPHp9Do/items/7QRA59ZE,urihttp//zotero.org/users/local/zTPHp9Do/items/7QRA59ZE,itemDataid174,typearticle-journal,titleAsthma-COPD overlap,container-titleChest,page7-8,volume149,issue1,authorfamilyBarnes,givenPeter J.,issueddate-parts2016,schemahttps//github.com/citation-style-language/schema/raw/master/csl-citation.json (Barnes, 2016).

Li, Y., Li, G. P. (2016). Oxidative stress in asthma a distinct clinical and pathologic feature.Journal of biological regulators and homeostatic agents,30(4), 1053-1057.

Both the authors Y Li and GP Li, after substantial research found out that Asthma is a critical airway inflammation. Their main view was if oxidative stress is a distinctive medical and pathologic characteristic of asthma. After reviewing various articles on asthma-related oxidative stress, they found out that contact with airborne allergens such as dust and pollen, not only triggered the adaptive immune response, but also were a cause of oxidative stress harm in the airway. They believe that airborne particles are a substantial environment pollutant. They need to be studied further as they are causing progress in the asthma-related oxidative stress in the airways. Regardless of the relation of oxidative stress with the asthma degree, this area further needs to be studied. However, antioxidant treatment has proven to be useful for oxidative stress related to asthma ADDIN ZOTERO_ITEM CSL_CITATION citationIDa1123plgl9e,propertiesformattedCitation(Li Li, 2016),plainCitation(Li Li, 2016),citationItemsid175,urishttp//zotero.org/users/local/zTPHp9Do/items/NCICAIM7,urihttp//zotero.org/users/local/zTPHp9Do/items/NCICAIM7,itemDataid175,typearticle-journal,titleOxidative stress in asthma a distinct clinical and pathologic feature,container-titleJournal of biological regulators and homeostatic agents,page1053-1057,volume30,issue4,authorfamilyLi,givenY.,familyLi,givenG. P.,issueddate-parts2016,schemahttps//github.com/citation-style-language/schema/raw/master/csl-citation.json (Li Li, 2016).

Lambrecht, B. N., Persson, E. K., Hammad, H. (2017). Myeloid Cells in Asthma.Microbiology spectrum,5(1).

The authors of the article after critical research found out that Asthma is indeed a varied long-lasting inflammatory disorder of the airway. It came to them as a surprise that those myeloid cells have a play in the pathogenesis. The antigen presenting cells are responsible for the recognition of the various allergens and pollutants. They initiate the immune response by traveling to the lymph nodes. However, the majority of myeloid cells have a good response to inhaling steroids. Having said that, the targeted therapy for these cells can eliminate only a single population of these cells, but with the aid of new tools, we will be entirely able to understand the reality of myeloid cells in chronic asthma in regards to humans ADDIN ZOTERO_ITEM CSL_CITATION citationIDa7p81ku540,propertiesformattedCitation(Lambrecht, Persson, Hammad, 2017),plainCitation(Lambrecht, Persson, Hammad, 2017),citationItemsid173,urishttp//zotero.org/users/local/zTPHp9Do/items/GMEYW866,urihttp//zotero.org/users/local/zTPHp9Do/items/GMEYW866,itemDataid173,typearticle-journal,titleMyeloid Cells in Asthma.,container-titleMicrobiology spectrum,volume5,issue1,authorfamilyLambrecht,givenBart N.,familyPersson,givenEmma K.,familyHammad,givenHamida,issueddate-parts2017,schemahttps//github.com/citation-style-language/schema/raw/master/csl-citation.json (Lambrecht, Persson, Hammad, 2017).

References

Barnes, P. J. (2016). Asthma-COPD overlap.Chest,149(1), 7-8.

Li, Y., Li, G. P. (2016). Oxidative stress in asthma a distinct clinical and pathologic feature.Journal of biological regulators and homeostatic agents,30(4), 1053-1057.

Lambrecht, B. N., Persson, E. K., Hammad, H. (2017). Myeloid Cells in Asthma.Microbiology spectrum,5(1).

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HEALTHCARE AND NURSING

1

Running head HEALTHCARE AND NURSING

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